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rabbit anti human cd68 mab multimabtm  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc rabbit anti human cd68 mab multimabtm
    A.U. arbitrary unit, RT-qPCR reverse transcription quantitative polymerase chain reaction, WT THP1 WT-fortilin cells, KO THP1 KO-fortilin cells, oxLDL oxidized low density lipoprotein, αSMA alpha smooth muscle cell actin, <t>CD68</t> Cluster of Differentiation 68, TCE, 2,2,2-trichloroethanol total protein staining, GAPDH glyceraldehyde-3-phosphate dehydrogenase, phMΦ primary human macrophages, CTL control, phMΦ transduced by control short hairpin RNA (shRNA control ) lentiviral particles, KD knockdown, phMΦ transduced by shRNA fortilin lentiviral particles. a Expression levels of fortilin in WT and KO cells with and without oxLDL stimulation. b – f Status of expression of VSMC markers (αSMA, SM22α, CNN1) ( b – d ) and MФ markers (Mac2, CD68) ( e , f ) in WT and KO cells as analyzed by RT-qPCR (n = 3, 3). g Western blot analysis of VSMC and MΦ markers in WT and KO cells at their baselines and upon oxLDL stimulation. h Successful KD of fortilin using shRNA fortilin lentivirus particles in phMΦ. Status of expression of VSMC markers ( i – k ) and MΦ markers ( l , m ) in CTL and KD phMΦ cells as analyzed by RT-qPCR (n = 3, 3). Data are expressed as mean ± s.d., P values determined by one-way ANOVA with Tukey multiple comparisons ( a – f , h – m ) are shown. The findings in ( g ) were confirmed in three independent experiments.
    Rabbit Anti Human Cd68 Mab Multimabtm, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 10 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rabbit+anti+human+cd68+mab+multimabtm/CD68+MultiMab+Rabbit+mAb+mix/pmc12246171-307-152-156
    Average 94 stars, based on 10 article reviews
    rabbit anti human cd68 mab multimabtm - by Bioz Stars, 2026-11
    94/100 stars

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    1) Product Images from "Fortilin deficiency induces anti-atherosclerotic phenotypes in macrophages and protects hypercholesterolemic mice against atherosclerosis"

    Article Title: Fortilin deficiency induces anti-atherosclerotic phenotypes in macrophages and protects hypercholesterolemic mice against atherosclerosis

    Journal: Communications Biology

    doi: 10.1038/s42003-025-08425-w

    A.U. arbitrary unit, RT-qPCR reverse transcription quantitative polymerase chain reaction, WT THP1 WT-fortilin cells, KO THP1 KO-fortilin cells, oxLDL oxidized low density lipoprotein, αSMA alpha smooth muscle cell actin, CD68 Cluster of Differentiation 68, TCE, 2,2,2-trichloroethanol total protein staining, GAPDH glyceraldehyde-3-phosphate dehydrogenase, phMΦ primary human macrophages, CTL control, phMΦ transduced by control short hairpin RNA (shRNA control ) lentiviral particles, KD knockdown, phMΦ transduced by shRNA fortilin lentiviral particles. a Expression levels of fortilin in WT and KO cells with and without oxLDL stimulation. b – f Status of expression of VSMC markers (αSMA, SM22α, CNN1) ( b – d ) and MФ markers (Mac2, CD68) ( e , f ) in WT and KO cells as analyzed by RT-qPCR (n = 3, 3). g Western blot analysis of VSMC and MΦ markers in WT and KO cells at their baselines and upon oxLDL stimulation. h Successful KD of fortilin using shRNA fortilin lentivirus particles in phMΦ. Status of expression of VSMC markers ( i – k ) and MΦ markers ( l , m ) in CTL and KD phMΦ cells as analyzed by RT-qPCR (n = 3, 3). Data are expressed as mean ± s.d., P values determined by one-way ANOVA with Tukey multiple comparisons ( a – f , h – m ) are shown. The findings in ( g ) were confirmed in three independent experiments.
    Figure Legend Snippet: A.U. arbitrary unit, RT-qPCR reverse transcription quantitative polymerase chain reaction, WT THP1 WT-fortilin cells, KO THP1 KO-fortilin cells, oxLDL oxidized low density lipoprotein, αSMA alpha smooth muscle cell actin, CD68 Cluster of Differentiation 68, TCE, 2,2,2-trichloroethanol total protein staining, GAPDH glyceraldehyde-3-phosphate dehydrogenase, phMΦ primary human macrophages, CTL control, phMΦ transduced by control short hairpin RNA (shRNA control ) lentiviral particles, KD knockdown, phMΦ transduced by shRNA fortilin lentiviral particles. a Expression levels of fortilin in WT and KO cells with and without oxLDL stimulation. b – f Status of expression of VSMC markers (αSMA, SM22α, CNN1) ( b – d ) and MФ markers (Mac2, CD68) ( e , f ) in WT and KO cells as analyzed by RT-qPCR (n = 3, 3). g Western blot analysis of VSMC and MΦ markers in WT and KO cells at their baselines and upon oxLDL stimulation. h Successful KD of fortilin using shRNA fortilin lentivirus particles in phMΦ. Status of expression of VSMC markers ( i – k ) and MΦ markers ( l , m ) in CTL and KD phMΦ cells as analyzed by RT-qPCR (n = 3, 3). Data are expressed as mean ± s.d., P values determined by one-way ANOVA with Tukey multiple comparisons ( a – f , h – m ) are shown. The findings in ( g ) were confirmed in three independent experiments.

    Techniques Used: Quantitative RT-PCR, Reverse Transcription, Real-time Polymerase Chain Reaction, Staining, Control, shRNA, Knockdown, Expressing, Western Blot

    Related Articles

    Recombinant:

    Article Title: Fortilin deficiency induces anti-atherosclerotic phenotypes in macrophages and protects hypercholesterolemic mice against atherosclerosis.
    Article Snippet: .. The following primary antibodies were used: • Rabbit anti-fortilin (MBL International,Woburn,MA,USA;Catalog #: PM017; 1:1,000 dilution; used for the experiments depicted in Fig. 1c); • Rabbit anti-fortilin (Abcam, Waltham, MA, USA; Clone EPR5540, ab133568; 1:2,000 dilution; Figs. 5g, S4a); • Mouse anti-GAPDH(Fitzgerald,Acton,MA,USA;Clone: 10R-G109a; 1:10,000 dilution; Fig. 1c); mouse anti-GAPDH (Santa Cruz Biotechnology, Inc. Dallas, TX, USA; Clone: 6C5; Catalog #: sc-32233; 1:1,000 dilution; Figs. 5g, S6e); • Rabbit anti-human calponin-1 (CNN1)mAb (Cell Signaling Technology (CST), Danvers, MA, USA; Clone D8L2T, Catalog #: 17819; 1:1,000 dilution; Fig. 5g); • Recombinant rabbit anti-human α-smooth muscle actin (SMA) antibody (Abcam, clone EPR5368, Catalog #: ab124964; 1:1,000 dilution; Fig. 5g); • Rabbit anti-human SM22α (Transgelin/TAGLN) polyclonal Ab (CST, Catalog #: 40471; 1:1,000 dilution; Fig. 5g); • Rabbit anti-human TGF-β1 polyclonal Ab (Abcam, Catalog #: ab92486; 1:1,000 dilution; Figs. 5g, S6e); • Rabbit anti-Mac2 (Galectin-3/LGALS3) mAb (CST, Clone: D4I2R, Catalog #: 8785; 1:1,000 dilution; Fig. 5g); • Rabbit anti-human CD68 mAb MultiMabTM(CST, multiple clones, Catalog #: 86985; 1:1,000 dilution; Fig. 5g). .. For the quantification of CD36 expression in THP1KO-fortilin and THP1WT-fortilin cells, we used JESSTM, a highly quantitative capillary-based western blot system (ProteinSimple®, San Jose, CA, USA)24.

    Article Title: Fortilin deficiency induces anti-atherosclerotic phenotypes in macrophages and protects hypercholesterolemic mice against atherosclerosis
    Article Snippet: .. The following primary antibodies were used: Rabbit anti-fortilin (MBL International, Woburn, MA, USA; Catalog #: PM017; 1:1,000 dilution; used for the experiments depicted in Fig. ); Rabbit anti-fortilin (Abcam, Waltham, MA, USA; Clone EPR5540, ab133568; 1:2,000 dilution; Figs. , ); Mouse anti-GAPDH (Fitzgerald, Acton, MA, USA; Clone: 10R-G109a; 1:10,000 dilution; Fig. ); mouse anti-GAPDH (Santa Cruz Biotechnology, Inc. Dallas, TX, USA; Clone: 6C5; Catalog #: sc-32233; 1:1,000 dilution; Figs. , ); Rabbit anti-human calponin-1 (CNN1) mAb (Cell Signaling Technology (CST), Danvers, MA, USA; Clone D8L2T, Catalog #: 17819; 1:1,000 dilution; Fig. ); Recombinant rabbit anti-human α-smooth muscle actin (SMA) antibody (Abcam, clone EPR5368, Catalog #: ab124964; 1:1,000 dilution; Fig. ); Rabbit anti-human SM22α (Transgelin/TAGLN) polyclonal Ab (CST, Catalog #: 40471; 1:1,000 dilution; Fig. ); Rabbit anti-human TGF-β1 polyclonal Ab (Abcam, Catalog #: ab92486; 1:1,000 dilution; Figs. , ); Rabbit anti-Mac2 (Galectin-3/LGALS3) mAb (CST, Clone: D4I2R, Catalog #: 8785; 1:1,000 dilution; Fig. ); Rabbit anti-human CD68 mAb MultiMabTM(CST, multiple clones, Catalog #: 86985; 1:1,000 dilution; Fig. ). .. For the quantification of CD36 expression in THP1 KO-fortilin and THP1 WT-fortilin cells, we used JESSTM, a highly quantitative capillary-based western blot system (ProteinSimple®, San Jose, CA, USA) .

    Clone Assay:

    Article Title: Fortilin deficiency induces anti-atherosclerotic phenotypes in macrophages and protects hypercholesterolemic mice against atherosclerosis.
    Article Snippet: .. The following primary antibodies were used: • Rabbit anti-fortilin (MBL International,Woburn,MA,USA;Catalog #: PM017; 1:1,000 dilution; used for the experiments depicted in Fig. 1c); • Rabbit anti-fortilin (Abcam, Waltham, MA, USA; Clone EPR5540, ab133568; 1:2,000 dilution; Figs. 5g, S4a); • Mouse anti-GAPDH(Fitzgerald,Acton,MA,USA;Clone: 10R-G109a; 1:10,000 dilution; Fig. 1c); mouse anti-GAPDH (Santa Cruz Biotechnology, Inc. Dallas, TX, USA; Clone: 6C5; Catalog #: sc-32233; 1:1,000 dilution; Figs. 5g, S6e); • Rabbit anti-human calponin-1 (CNN1)mAb (Cell Signaling Technology (CST), Danvers, MA, USA; Clone D8L2T, Catalog #: 17819; 1:1,000 dilution; Fig. 5g); • Recombinant rabbit anti-human α-smooth muscle actin (SMA) antibody (Abcam, clone EPR5368, Catalog #: ab124964; 1:1,000 dilution; Fig. 5g); • Rabbit anti-human SM22α (Transgelin/TAGLN) polyclonal Ab (CST, Catalog #: 40471; 1:1,000 dilution; Fig. 5g); • Rabbit anti-human TGF-β1 polyclonal Ab (Abcam, Catalog #: ab92486; 1:1,000 dilution; Figs. 5g, S6e); • Rabbit anti-Mac2 (Galectin-3/LGALS3) mAb (CST, Clone: D4I2R, Catalog #: 8785; 1:1,000 dilution; Fig. 5g); • Rabbit anti-human CD68 mAb MultiMabTM(CST, multiple clones, Catalog #: 86985; 1:1,000 dilution; Fig. 5g). .. For the quantification of CD36 expression in THP1KO-fortilin and THP1WT-fortilin cells, we used JESSTM, a highly quantitative capillary-based western blot system (ProteinSimple®, San Jose, CA, USA)24.

    Article Title: Fortilin deficiency induces anti-atherosclerotic phenotypes in macrophages and protects hypercholesterolemic mice against atherosclerosis
    Article Snippet: .. The following primary antibodies were used: Rabbit anti-fortilin (MBL International, Woburn, MA, USA; Catalog #: PM017; 1:1,000 dilution; used for the experiments depicted in Fig. ); Rabbit anti-fortilin (Abcam, Waltham, MA, USA; Clone EPR5540, ab133568; 1:2,000 dilution; Figs. , ); Mouse anti-GAPDH (Fitzgerald, Acton, MA, USA; Clone: 10R-G109a; 1:10,000 dilution; Fig. ); mouse anti-GAPDH (Santa Cruz Biotechnology, Inc. Dallas, TX, USA; Clone: 6C5; Catalog #: sc-32233; 1:1,000 dilution; Figs. , ); Rabbit anti-human calponin-1 (CNN1) mAb (Cell Signaling Technology (CST), Danvers, MA, USA; Clone D8L2T, Catalog #: 17819; 1:1,000 dilution; Fig. ); Recombinant rabbit anti-human α-smooth muscle actin (SMA) antibody (Abcam, clone EPR5368, Catalog #: ab124964; 1:1,000 dilution; Fig. ); Rabbit anti-human SM22α (Transgelin/TAGLN) polyclonal Ab (CST, Catalog #: 40471; 1:1,000 dilution; Fig. ); Rabbit anti-human TGF-β1 polyclonal Ab (Abcam, Catalog #: ab92486; 1:1,000 dilution; Figs. , ); Rabbit anti-Mac2 (Galectin-3/LGALS3) mAb (CST, Clone: D4I2R, Catalog #: 8785; 1:1,000 dilution; Fig. ); Rabbit anti-human CD68 mAb MultiMabTM(CST, multiple clones, Catalog #: 86985; 1:1,000 dilution; Fig. ). .. For the quantification of CD36 expression in THP1 KO-fortilin and THP1 WT-fortilin cells, we used JESSTM, a highly quantitative capillary-based western blot system (ProteinSimple®, San Jose, CA, USA) .



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    rabbit anti human cd68 mab multimabtm  (Cell Signaling Technology Inc)
    94
    Cell Signaling Technology Inc rabbit anti human cd68 mab multimabtm
    A.U. arbitrary unit, RT-qPCR reverse transcription quantitative polymerase chain reaction, WT THP1 WT-fortilin cells, KO THP1 KO-fortilin cells, oxLDL oxidized low density lipoprotein, αSMA alpha smooth muscle cell actin, <t>CD68</t> Cluster of Differentiation 68, TCE, 2,2,2-trichloroethanol total protein staining, GAPDH glyceraldehyde-3-phosphate dehydrogenase, phMΦ primary human macrophages, CTL control, phMΦ transduced by control short hairpin RNA (shRNA control ) lentiviral particles, KD knockdown, phMΦ transduced by shRNA fortilin lentiviral particles. a Expression levels of fortilin in WT and KO cells with and without oxLDL stimulation. b – f Status of expression of VSMC markers (αSMA, SM22α, CNN1) ( b – d ) and MФ markers (Mac2, CD68) ( e , f ) in WT and KO cells as analyzed by RT-qPCR (n = 3, 3). g Western blot analysis of VSMC and MΦ markers in WT and KO cells at their baselines and upon oxLDL stimulation. h Successful KD of fortilin using shRNA fortilin lentivirus particles in phMΦ. Status of expression of VSMC markers ( i – k ) and MΦ markers ( l , m ) in CTL and KD phMΦ cells as analyzed by RT-qPCR (n = 3, 3). Data are expressed as mean ± s.d., P values determined by one-way ANOVA with Tukey multiple comparisons ( a – f , h – m ) are shown. The findings in ( g ) were confirmed in three independent experiments.
    Rabbit Anti Human Cd68 Mab Multimabtm, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rabbit+anti+human+cd68+mab+multimabtm/CD68+MultiMab+Rabbit+mAb+mix/pmc12246171-307-152-156
    Average 94 stars, based on 1 article reviews
    rabbit anti human cd68 mab multimabtm - by Bioz Stars, 2026-11
    94/100 stars
    		  Buy from Supplier

    Image Search Results


    A.U. arbitrary unit, RT-qPCR reverse transcription quantitative polymerase chain reaction, WT THP1 WT-fortilin cells, KO THP1 KO-fortilin cells, oxLDL oxidized low density lipoprotein, αSMA alpha smooth muscle cell actin, CD68 Cluster of Differentiation 68, TCE, 2,2,2-trichloroethanol total protein staining, GAPDH glyceraldehyde-3-phosphate dehydrogenase, phMΦ primary human macrophages, CTL control, phMΦ transduced by control short hairpin RNA (shRNA control ) lentiviral particles, KD knockdown, phMΦ transduced by shRNA fortilin lentiviral particles. a Expression levels of fortilin in WT and KO cells with and without oxLDL stimulation. b – f Status of expression of VSMC markers (αSMA, SM22α, CNN1) ( b – d ) and MФ markers (Mac2, CD68) ( e , f ) in WT and KO cells as analyzed by RT-qPCR (n = 3, 3). g Western blot analysis of VSMC and MΦ markers in WT and KO cells at their baselines and upon oxLDL stimulation. h Successful KD of fortilin using shRNA fortilin lentivirus particles in phMΦ. Status of expression of VSMC markers ( i – k ) and MΦ markers ( l , m ) in CTL and KD phMΦ cells as analyzed by RT-qPCR (n = 3, 3). Data are expressed as mean ± s.d., P values determined by one-way ANOVA with Tukey multiple comparisons ( a – f , h – m ) are shown. The findings in ( g ) were confirmed in three independent experiments.

    Journal: Communications Biology

    Article Title: Fortilin deficiency induces anti-atherosclerotic phenotypes in macrophages and protects hypercholesterolemic mice against atherosclerosis

    doi: 10.1038/s42003-025-08425-w

    Figure Lengend Snippet: A.U. arbitrary unit, RT-qPCR reverse transcription quantitative polymerase chain reaction, WT THP1 WT-fortilin cells, KO THP1 KO-fortilin cells, oxLDL oxidized low density lipoprotein, αSMA alpha smooth muscle cell actin, CD68 Cluster of Differentiation 68, TCE, 2,2,2-trichloroethanol total protein staining, GAPDH glyceraldehyde-3-phosphate dehydrogenase, phMΦ primary human macrophages, CTL control, phMΦ transduced by control short hairpin RNA (shRNA control ) lentiviral particles, KD knockdown, phMΦ transduced by shRNA fortilin lentiviral particles. a Expression levels of fortilin in WT and KO cells with and without oxLDL stimulation. b – f Status of expression of VSMC markers (αSMA, SM22α, CNN1) ( b – d ) and MФ markers (Mac2, CD68) ( e , f ) in WT and KO cells as analyzed by RT-qPCR (n = 3, 3). g Western blot analysis of VSMC and MΦ markers in WT and KO cells at their baselines and upon oxLDL stimulation. h Successful KD of fortilin using shRNA fortilin lentivirus particles in phMΦ. Status of expression of VSMC markers ( i – k ) and MΦ markers ( l , m ) in CTL and KD phMΦ cells as analyzed by RT-qPCR (n = 3, 3). Data are expressed as mean ± s.d., P values determined by one-way ANOVA with Tukey multiple comparisons ( a – f , h – m ) are shown. The findings in ( g ) were confirmed in three independent experiments.

    Article Snippet: The following primary antibodies were used: Rabbit anti-fortilin (MBL International, Woburn, MA, USA; Catalog #: PM017; 1:1,000 dilution; used for the experiments depicted in Fig. ); Rabbit anti-fortilin (Abcam, Waltham, MA, USA; Clone EPR5540, ab133568; 1:2,000 dilution; Figs. , ); Mouse anti-GAPDH (Fitzgerald, Acton, MA, USA; Clone: 10R-G109a; 1:10,000 dilution; Fig. ); mouse anti-GAPDH (Santa Cruz Biotechnology, Inc. Dallas, TX, USA; Clone: 6C5; Catalog #: sc-32233; 1:1,000 dilution; Figs. , ); Rabbit anti-human calponin-1 (CNN1) mAb (Cell Signaling Technology (CST), Danvers, MA, USA; Clone D8L2T, Catalog #: 17819; 1:1,000 dilution; Fig. ); Recombinant rabbit anti-human α-smooth muscle actin (SMA) antibody (Abcam, clone EPR5368, Catalog #: ab124964; 1:1,000 dilution; Fig. ); Rabbit anti-human SM22α (Transgelin/TAGLN) polyclonal Ab (CST, Catalog #: 40471; 1:1,000 dilution; Fig. ); Rabbit anti-human TGF-β1 polyclonal Ab (Abcam, Catalog #: ab92486; 1:1,000 dilution; Figs. , ); Rabbit anti-Mac2 (Galectin-3/LGALS3) mAb (CST, Clone: D4I2R, Catalog #: 8785; 1:1,000 dilution; Fig. ); Rabbit anti-human CD68 mAb MultiMabTM(CST, multiple clones, Catalog #: 86985; 1:1,000 dilution; Fig. ).

    Techniques: Quantitative RT-PCR, Reverse Transcription, Real-time Polymerase Chain Reaction, Staining, Control, shRNA, Knockdown, Expressing, Western Blot